Transgenic mice with inducible cardiomyocyte-specific expression of a wild type form of the human angiotensin II (Ang II) type-1 receptor (hAT1R) showed strong cardiomyocyte-specific LacZ activation in both neonatal and adult hearts, and pulmonary veins when induced. When adults were given dox, the receptor expression was repressed. (J:162208)