这个转基因表达了一种缺陷型的 Akt1 基因,其中关键的 ATP 结合位点被突变(K179M),它在mouse alpha-肌动蛋白重链的调控下表达。(来源:J:75780)
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This transgene expresses a kinase deficient mouse Akt1, in which the critical ATP binding site was mutated (K179M), under the control of the mouse alpha-myosin heavy chain. (J:75780)