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The CAG promoter was used to drive expression of a construct composed of a floxed EGFP with a strong termination signal followed by a myc tagged dominant active form of GLI2 lacking the N terminal repressor domain (GLI2deltaN). In the absence of cre, EGFPis expressed and downstream GLI2 expression is prevented. Cre expression causes the excision of EGFP and its termination signal thus permitting GLI2 expression. (J:116111, J:154130)
Basic Information
Allele
Strain of Origin
Allele Type
Mutation
Inheritance
Gene Expression
Related Disease
Reference
Phenotypes
Legend：
hm: homozygous
ht: heterozygous
cn: conditional genotype
cx: complex: > 1 genome feature
tg: involves transgenes
ot: other: hemizygous, indeterminate,...
(F): Female
(M): Male
phenotype observed
N: normal phenotype
(#): related diseases count
Phenotypes：
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| Phenotypes |
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References Literature
Title
PMID
Journal
Year
IF
